Integumentary System Assessment

Skin: Inspection

Inspection of the skin can be performed with the client sitting upright on the exam table or lying in a supine position. If you are doing a focused assessment, position the client so that you have the best visibility of the affected area. Use a bright light or a pen light if needed. Shadows and dark lighting may alter your visualization of the affected area and provide information about the elevation of a lump. 

If you are doing a complete assessment, use a systematic approach and proceed to ensure comprehensiveness. If the client has identified a specific concern, inspect that area first. If you are doing a complete assessment when the client has no specific concerns, inspect the face, arms/hands, back, abdomen, chest, and legs/feet. Always inspect the anterior and posterior side; for example, for the arms, ask the client to raise their hands/arms in front of them with their palms facing down and then turn palms up so that you can inspect both sides. Similarly, for the legs, inspect the anterior and then have the client roll over so that you can inspect the posterior. You may need to seek assistance to reposition a client that is immobile or has difficulty moving. 

Keep in mind that the best control for comparison is the client’s own body, so always compare bilaterally

Clinical Tip

Commonly overlooked areas for skin inspection include behind the knees, in , and between digits (fingers and toes). Other important areas to check are bony prominences (e.g., hips, spine, ankles), especially if the client has mobility issues. If the client is unable to reach these areas, encouraging family members to assist with checks can be helpful.

Skin inspection involves the following steps:

  1. Inspect the skin for colour using both a generalized and localized approach. To visualize general colour, step back and take note of their general appearance; for a localized approach, inspect specific regions. Mucous membranes, palms, sclera, and regions around the mouth can all be telltale signs for centralized colour variations. Always examine distal extremities for discolouration. Table 4 lists common colour variations. Skin colour varies widely from dark to light shades including black, brown, yellow, and white shades. These colours are affected by many factors. The biggest factor is how much is produced by the body’s melanocytes; this also affects hair and eye colour. Additionally, the circulating hemoglobin affects skin colour and the underlying connective tissue.
    • Normally, skin colour is evenly distributed with no variations; clients with darker skin tones will have lighter coloured palms and soles of feet. You may observe birthmarks, which are visible upon birth and include a wide array of sizes and colours (brown, black, blue, red, pink and purple). Most are flat and some fade over time. You should document them describing their location and description particularly since some can look like bruising.  
    • If you notice variation in skin colour, note the location, colour, and other characteristics of the discolouration.

Table 4: Colour variations.

 

Colour Variation 

 

Skin Tone Variations and Clinical Tip

Pallor is a lightening of the skin compared to the client’s typical complexion. It is commonly due to a lack of oxygen-rich blood near the surface of the skin. 

Among clients with darker skin tones, pallor may present as ashen (grey) colouring. Pallor may also present as a yellowish colour among persons with browner skin tones. Among clients with lighter skin tones, the skin appears paler and less pink. To check for pallor, look at mucus membranes, nail beds, conjunctiva, and/or palms of hands. These are all highly vascularized regions that tend to be a pink colour. With pallor, they look pale. 

Cyanosis is a bluish/whitish/greyish discolouration of the skin, usually due to a lack of oxygen in the blood. Peripheral cyanosis occurs in the distal extremities, while central cyanosis is generalized in the trunk and head of the body. 

With darker skin tones, cyanosis may present as a whitish/greyish colour. For clients with yellow undertones to their skin, cyanosis may be a greenish/greyish colour. For persons with lighter skin tones, cyanosis is generally a bluish/purple hue. Centralized cyanosis is generally most pronounced around the mouth (tongue, lips, oral cavity) and mucous membranes. Cheeks, nose and ears are also good landmarks to check for centralized cyanosis as these are highly vascular regions with thin skin.

Erythema is a reddening/darkening of the skin, typically due to increased blood flow to the capillaries. 

With darker skin tones, erythema may appear reddish/purple in colour. With very dark skin tones, it may be difficult to visualize. With lighter skin tones, erythema is typically dark pink to red. As you check for erythema, look for other signs like inflammation and warmth. 

Brawny is a brown-reddish discolouration, typically associated with venous insufficiency. Red blood cells accumulate in the interstitial spaces and can cause from the blood leaking out of capillaries. 

With darker skin tones, brawny may appear even darker than the rest of the skin, with a brown-reddish hue. With lighter skin tones, the skin is a dark brown-reddish colour. Brawny generally appears in lower extremities and regions of the body that have pooling due to venous insufficiency. 

Jaundice is a yellowing discolouration of skin, sclera, and mucous membranes. It is typically brought on by a buildup of and breakdown of red blood cells in the body.

A person with jaundice skin and eyes.

(Attribution: Photo by By James Heilman, MD – Own work, CC BY 3.0, https://commons.wikimedia.org/w/index.php?curid=9389660)

In persons with darker skin tones, discolouration may be subtle or go unnoticed but visible in the sclera as shown in image below. In persons with lighter skin tones, jaundice can appear yellow or even orange in colour. Jaundice is typically confirmed by blood test or urinalysis. 

A person's face that has jaundice eyes and skin.

 

 

 

 

 

 

(Attribution: Photo by Unknown author – CDC / Dr. Thomas F. Sellers / Emory University, Public Domain, https://commons.wikimedia.org/w/index.php?curid=4394119)

Vitiligo is a condition where the skin loses pigmentation in certain regions (patchy).

Partially lost pigmentation.

Image is free to use for non-commercial purposes from:https://www.atlasdermatologico.com.br/index.jsf

Vitiligo can affect persons with all skin tones, but it is more visible among persons with darker skin tones due to contrast. Common regions for vitiligo are near the mouth and eyes or on the fingers, wrists, armpits, or groin. 

  1. Inspect the skin for nevi (moles). If the client has expressed concern about specific moles, check them first. Otherwise, inspect the face, arms/hands, back, chest, abdomen, and legs/feet for the presence of nevi. If you observe any, use the ABCDE mole screening mnemonic (Table 5 and Figure 4); this is vital to screen for melanoma, a life-threatening skin cancer in which early detection is essential (Rigel et al., 2005). Most moles are benign, but they can become malignant: people with more than 50 moles, light-coloured skin, regular sun exposure without sunscreen, and/or a family history of melanoma are at increased risk for melanoma.
    • Normally, nevi are smaller than 6 mm in diameter, round, smooth surfaced, with distinct/round edges. They can be elevated or not, pink, tan, brown, or dark brown. Generally, persons with darker skin tones have darker moles and persons with lighter skin tone have lighter moles. See Figure 5 for examples of what is considered normal nevi.
    • Abnormal moles are often larger than 6 mm with variation in colours and asymmetrical with irregular borders. Note any atypical moles including the location and description. 

Four pictures showing an asymmetrical nevi, a mole with border irregularity, a mole with colour variation, a large mole.

Figure 4: Abnormal nevi/moles. (Credit: Courtesy of Skin Cancer Foundation/National Cancer Institute License: Public domain. Source: Left to right: http://visualsonline.cancer.gov/details.cfm?imageid=2362; http://visualsonline.cancer.gov/details.cfm?imageid=2363; http://visualsonline.cancer.gov/details.cfm?imageid=2364; http://visualsonline.cancer.gov/details.cfm?imageid=2184).

Several flat brown nevi on the back with two large, slightly raised brown nevi with regular borders and symmetrical. A dark brown, raised nevi that is symmetrical with regular borders.

Figure 5: Examples of normal nevi/moles. 

Table 5: ABCDE mole screening. (Adapted from Rigel et al., 2005. See the American Academy of Dermatology Association about the history of the mnemonic: https://www.aad.org/public/diseases/skin-cancer/find/at-risk/abcdes)

 

Screening Characteristic

 

Warning Sign 

 

A – Asymmetry

 

Moles that are asymmetrical/irregular in shape (one side does not match the other).

 

B – Border irregularity

 

Mole borders that are irregular or jagged in appearance.

 

C – Colour

 

Moles that have more than one colour within the mole (mixture of colours like tan, brown, black, red/pink) or changes colour (white, red, or blue) or a dark mole that does not match other moles on the client’s body (dark black). 

 

D – Diameter greater than 6 mm

 

Moles that are larger than 6 mm (pea-size) or moles that start small and grow larger than 6 mm. 

 

E – Evolving

 

Moles that have changed/evolved in terms of bleeding, pain, size, colour, shape, texture (crusting), elevation, or itching. 

Clinical Tip

ABCDE is a useful mnemonic for screening moles and is easily teachable to most clients. Teach-back is an effective technique: this involves presenting the ABCDE mnemonic to the client and then having them “teach” it back to you. This method helps with memory retention; fill in any gaps you may have overlooked as you taught them. Ask them to screen one of their moles based on the mnemonic; it may be appropriate to take a picture for comparison purposes.

  1. Inspect for skin integrity including whether the skin is intact. Look for the presence of , , , or other damage that can disrupt the normal pattern of the skin. You will learn to recognize the signs of skin breakdown and who is at risk for impaired skin integrity. An important tool to assess skin integrity is the Braden Scale, which is commonly used in many healthcare settings and is especially useful for hospitalized clients and those with restricted mobility. 
    • Skin is normally intact with no lesions, ulcerations, erosions, and contusions. 
    • If you observe any areas where the skin is not intact, note the location and describe the area. Pressure injuries are classified into stages, as shown in Table 6Figure 6 presents an example of staging.

Braden Scale

The Braden Scale is an established tool used to screen and assess for risk of developing pressure sores (Bergstrom et al., 1987; Braden, 2012). Clients at risk are screened weekly. Many factors can increase the risk of developing pressure injuries, including altered sensory perception, increased moisture, decreased activity, impaired mobility, inadequate nutrition, and issues with friction and shear (Open Resources for Nursing, n.d.). As Bergstrom and colleagues note, the Braden Scale is used to screen clients in six areas:

  • Sensory perception.
  • Skin moisture.
  • Activity.
  • Mobility.
  • Friction and shear.
  • Nutritional status.

Friction and shear is rated on a scale from 1–3; all other areas are rated on a scale of 1–4 (Open Resources for Nursing, n.d.). The scores for all six areas are totalled to indicate the client’s risk for developing a pressure injury based on the following ranges:

Mild risk: 15–18

Moderate risk: 13–14

High risk: 10–12

Severe risk: less than 9

(Bergstrom et al., 1998; Open Resources for Nursing, n.d.). 

Check out the Braden Scale at this link and assess your own risk, and then try using it with a client in your clinical setting:

https://www.clwk.ca/modules/Braden/Slide-1-14.html

Pressure Injury Staging

Classify a pressure injury according to Stages 1–4 or note it is unstageable as per the descriptions below and the example provided in Figure 6.

Table 6: Pressure injury staging. (Attribution: Adapted and modified from https://wtcs.pressbooks.pub/nursingfundamentals/)

 

Stage

 

Description 

 

Stage 1 pressure injury.

 

Intact skin with localized area of nonblanchable erythema where prolonged pressure has occurred. Nonblanchable erythema is a medical term used to describe a reddened skin area that does not turn white when pressed. 

 

Stage 2 pressure injury.

 

Partial-thickness loss of skin with exposed dermis. The wound bed is viable and may appear as an intact or ruptured blister.

 

Stage 3 pressure injury.

 

Full-thickness tissue loss in which subcutaneous tissue is visible, but cartilage, tendon, ligament, muscle, and bone are not. Depth of tissue damage varies by anatomical location. Undermining and tunneling may occur in Stage 3 and 4 pressure injuries. Undermining occurs when the tissue under the wound edge becomes eroded, resulting in a pocket beneath the skin. Tunneling refers to passageways underneath the skin surface that extend from a wound and can involve twists and turns.

Slough and eschar may also be present in Stage 3 and 4 pressure injuries. Slough is an inflammatory exudate that is usually light yellow, soft, and moist. Eschar is dark brown/black, dry, thick, and leathery dead tissue. If slough or eschar obscures the wound so that tissue loss cannot be assessed, the pressure injury is referred to as unstageable. In most wounds, slough and eschar must be removed by debridement for healing to occur.

 

Stage 4 pressure injury.

 

Full-thickness tissue loss with visible cartilage, tendon, ligament, muscle, or bone. Osteomyelitis (bone infection) may also be present. 

 

Unstageable (Stage X) pressure injury.

 

Full-thickness tissue loss in which the presence of slough or eschar are making it difficult to evaluate the extent of damage. If slough or eschar were to be removed, a Stage 3 or Stage 4 pressure injury would likely be revealed. 

Four pictures showing stage 1, stage 2, stage 3 and stage 4 of pressure injuries.

Figure 6: Example of pressure injury staging. (Attribution: Author Babagolzadeh, December 30, 2021, taken from https://commons.wikimedia.org/wiki/File:Wound_stage.jpg

This file is licensed under the Creative Commons Attribution-Share Alike 3.0 Unported license.)

  1. Inspect skin for other lesions such as cysts, blisters, macules, and wheals. Typically, lesions are associated with pain because they generally involve the epidermis and/or dermis which are both innervated (supplied by nerves). However, nerve damage may limit sensation and therefore pain to the region. Clients with nerve damage are at increased risk for secondary infections and lesions because they are not restricted by pain at the site. Lesions are usually categorized as primary (develop as a result of a pathological process and not modified by scratching or infection) or secondary (evolve from a primary lesion as a natural development or as a result of scratching or infection). Tables 7 and 8 provide more additional information about primary and secondary lesions. 
    • Normally, there are no lesions.
    • If lesions are present, describe the location and characteristics in detail, including size, colour, movability, borders, elevation, drainage, and pain levels. 

Table 7: Primary lesions. (Attribution: unless otherwise noted, lesion images adapted from https://commons.wikimedia.org/wiki/File:OSC_Microbio_21_01_LesionLine.jpg This file is licensed under the Creative Commons Attribution 4.0 International license).

 

Type

 

Example

 

Clinical consideration 

 

Abscess: Localized lump filled with pus.

 

Tooth abscess, peritonsillar abscess.

 

Pain is typically present.

 

Bulla: Fluid-filled blister.

 

Shingles, burns.

An animated image of vesicles and bulla.

(Attribution: Adapted photo by Madhero88 – Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/

index.php?curid=14546567)

 

Less than 5 mm in diameter.

 

Cyst: Encapsulated sac filled with fluid, semi-solid matter (such as dead skin cells), or gas; typically located in the upper layer of skin.

 

Dermoid, cutaneous, ganglion, sebaceous cysts.

An animated image of a cyst on the skin.

 

Firm masses can be cysts. 

 

Macule: Flat (non-palpable) spot typically discoloured (hyperpigmented or erythematous). 

 

Freckle, café au lait spot.

An animated image of a macule on the skin.

 

Less than 1 cm.

 

Nodule: Solid, elevated, palpable growth.

 

, some nevi.

An animated image of an exophutic nodule and endophytic nodule on the skin.

(Attribution: Photo by Madhero88 – Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/in

dex.php?curid=14546471)

 

1–2 cm in size.

 

Papule: Elevated, solid, palpable, circumscribed (with limits/bounded).

 

Elevated mole, mosquito bite.

An animated image of a papule on the skin.

 

Elevated less than 1 cm in diameter.

 

Plaque: Circumscribed, elevated, solid deposit.

 

Psoriasis, eczema, seborrheic dermatitis.

An animated image of a papule and plaque on the skin.

(Attribution: Adapted photo by Madhero88

– Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/i

ndex.php?curid=14546485)

 

Typically larger than 1 cm. 

 

Pustule: Pus-filled, circumscribed, elevated.

 

Pimple. 

An animated image of a pustule on the skin.

 

May have redness/swelling at the site of the pustule.

 

Tumour: Abnormal growth, palpable.

 

Lipoma, skin cancer.

 

Typically larger than nodules (> 2 cm).

 

Vesicle: Small, fluid-filled sacs, thin-walled.

 

Herpes simplex blister.

An animated image of a vesicle on the skin.

 

Usually appear in groups.

 

Wheal: Swollen, inflamed skin patch that itches or burns.

 

Hives.

An animated image of a wheal on the skin.

 

Generally 3 mm or larger. 

Table 8: Secondary lesions. (Attribution: unless otherwise noted, lesion images adapted from https://commons.wikimedia.org/wiki/File:OSC_Microbio_21_01_LesionLine.jpg This file is licensed under the Creative Commons Attribution 4.0 International license).

 

Type

 

Description

 

Atrophy

 

Thinning of the skin (sometimes shiny appearance), translucent, increased fragility. 

 

Crust

 

Accumulation of dried exudate and skin cells on the outer layer of the affected area (scab). 

An animated image of crust on the skin.

 

Erosion

 

Loss of parts of the epidermis.

An animated image of a fissure, erosion, and ulcer (left to right) on the skin.

(Attribution: Photo by Madhero88 – Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=14546561)

 

Excoriation 

 

Skin breakdown caused by repetitive scratching. 

 

Fissure

 

Crack or split of the outer layer of the skin. 

 

Keloid

 

Thick, raised patch of skin (scar tissue).

A raised, thick scar on stomach.

(Attribution: Photo by Htirgan – Own work, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=32782658)

 

Lichenification 

 

Hyperpigmentation and thickening of the skin. 

 

Scar

 

Fibrous, thick tissue, shiny appearance once lesion has healed.

 

Ulcer

 

Loss of parts of the tissue (crater-like), exposed with some healing formation.

An animated image of an ulcer on the skin.

  1. Note the findings
    • Normal findings might be documented as: “Skin integrity intact. Skin colour consistent throughout with no variations. No lesions or rashes noted. Nevus on back examined. Located 2 inches distal to the scapula, left side. Symmetrical with even borders, tan coloured, 3 mm in size and no changes noted by the client. No pain or sensation reported. Image taken and included in the chart.” 
    • Abnormal findings might be documented as: “Stage 1 pressure injury on thoracic spine 4 inches in length and 2 inches wide” 

Priorities of Care 

All abnormal findings should be documented and reported, but some findings are more urgent than others. For example, signs of cyanosis and pallor suggest possible issues with oxygenation, so you should conduct a primary survey, assess vital signs, and conduct a focused assessment on related systems including respiratory, cardiovascular, and peripheral vascular. A similar approach should be used when you observe mottled skin. This is a blotching and netlike discolouration that can appear as bluish, red, purple blotches, sometimes referred to as marbled. It is often associated with conditions that involved reduced blood flow and can be associated with peripheral vascular diseases, shock, and end-of-life, and sometimes cold environments. Always report signs of clinical deterioration immediately. For clients with a Stage 1 pressure injury, it is important to ensure good skin care and repositioning so that the client is not lying on a particular area for long periods of time. Medical intervention may be required for ulcers classified as Stage 2 and higher. Report any moles with warning signs to the physician or nurse practitioner, as the client may need a referral to a dermatologist and/or oncologist.

Knowledge Bite 

Burns are caused when the skin is damaged by intense heat, radiation, electricity, friction, or chemicals. This damage results in the death of skin cells. Loss of the skin’s protective layers can lead to massive loss of fluid, and makes burned skin extremely susceptible to infection.

Burns are classified by the degree of their severity

First-degree burn: 

  • Superficial burn affecting the epidermis. 
  • Mild sunburn is one example. 

Second-degree burn (see Figure 7):

  • Partial-thickness burn affecting the epidermis and a portion of the dermis. 
  • Results in swelling and a painful blistering of the skin. 

Third-degree burn: 

  • Full-thickness burn extends fully into the epidermis and dermis, destroying the tissue and affecting the nerve endings and sensory function. 

Fourth-degree burn: 

  • Deep full-thickness burn affecting the skin and underlying muscle, tendon, and bone. 

Third- and fourth-degree burns require immediate intervention. They are usually not as painful as second-degree burns because the nerve endings are damaged. Full-thickness burns require debridement (removal of dead skin) followed by grafting of the skin from an unaffected part of the body or from skin grown in tissue culture.

Swelling and blistering of the skin.

Figure 7: Second-degree burn.

(Attribution: Photo by Kronoman at English Wikipedia, CC BY-SA 3.0, https://commons.wikimedia.org/w/index.php?curid=26501619)

 

Activity: Check Your Understanding

References

Bergstrom, N., Braden, B., Kemp, M., Champagne, M., & Ruby, E. (1998). Predicting pressure ulcer risk: A multisite study of the predictive validity of the Braden Scale. Nursing Research, 47(5), 261-269.

Bergstrom, N., Braden, B., Laguzza, A., & Holman, V. (1987). The Braden Scale for predicting pressure sore risk. Nursing Research, 36(4), 205-210.

Braden, B. (2012). The Braden Scale for predicting pressure sore risk: Reflections after 25 years. Advances in Skin & Wound Care, 25(2), 61.

Open Resources for Nursing (n.d.). Chippewa Valley Technical College. https://wtcs.pressbooks.pub/nursingfundamentals/

Rigel, D., Friedman, R., Kopf, A., & Polsky, D. (2005). ABCDE–an evolving concept in the early detection of melanoma. Arch Dermatol, 141(8), 1032-1034.

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